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Curcumin for Amyloid Plaque Prevention
Curcumin binds amyloid-beta and reduces plaque in animal models, but human trials are small and hampered by poor bioavailability.
Overview
Verdict
Strong preclinical anti-amyloid data has not translated into convincing human outcomes.
How It Works
Dosing & Protocol
Typical dose
- Recommended dose
- No established dose — evidence is insufficient. Alzheimer's trials used 2-4 g/day curcumin or 180 mg/day of a bioavailable form, with no plaque benefit.
- Expected timeframe
- No benefit demonstrated over 6-18 months
Protocol
- dose
- Not established
- form
- Curcumin (Theracurmin 90 mg twice daily and 2-4 g/day plain curcumin were studied)
- steps
- Evidence is insufficient: randomised trials in Alzheimer's disease using 2-4 g/day curcumin, and 18-month trials of 180 mg/day Theracurmin, did not show reduced amyloid plaque or clinical benefit,Plain curcumin is poorly absorbed and reaches the brain in very low concentrations,Do not take curcumin expecting to prevent or clear amyloid plaque,For cognitive risk reduction, prioritise blood pressure, hearing, sleep, exercise and metabolic health
- timing
- n/a
- duration
- Trials ran 6-18 months
Evidence
What the studies say
Curcumin and cognition: a randomised, placebo-controlled, double-blind study of community-dwelling older adults
Curcumin produced small acute and chronic improvements in working memory and mood in healthy older adults.
Safety
Caveats
Insufficient evidence. Curcumin can increase bleeding risk with warfarin, clopidogrel or DOACs, may trigger gallbladder pain in gallstone disease, and rare idiosyncratic liver injury is reported. Avoid therapeutic doses in pregnancy.
Less likely to help if
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Individual results may vary. The statements on this website have not been evaluated by the Food and Drug Administration. Products and information are not intended to diagnose, treat, cure, or prevent any disease.