How Much Is Too Much? Exploring Pseudohyperaldosteronism in Glycyrrhizic Acid Toxicity From Chronic Licorice Root Consumption
Patel P, Aknouk M, Dawson A, Aya A, Kanukuntla A
Published in Cureus
Methodology
Case report and clinical review of glycyrrhizic acid toxicity presenting as pseudohyperaldosteronism after chronic licorice root consumption
Key Findings
Chronic licorice root intake produced hypertension, metabolic alkalosis and hypokalaemia consistent with pseudohyperaldosteronism.
Conclusions
Whole licorice root carries a real cardiovascular and electrolyte risk with sustained use.
Limitations
Single case; no dose-response data; not generalisable to deglycyrrhizinated products.
Supplements Studied
glycyrrhizic acid, the active component in licorice, can cause a metabolic syndrome presenting as pseudohyperaldosteronism
Study Details
Access Study
Disclosures
Funding
Not reported
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